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Fluoxetine further enhances NIX (BNIP3L)-mediated mitophagy in the hippocampus of CUMS mice [127, 128] and promotes autophagic flux and mitophagy in astrocytes via a p53-dependent mechanism, facilitating the clearance of damaged mitochondria [101]
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In a pre- and postnatal development study of SNAC exposure, an increase in gestation length, an increase in the number of stillbirths, and a decrease in pup viability were observed
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