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Acknowledgements This work was supported by the National Natural Science Foundation of China (grant # 81971256 [to HX]) and Natural Science Foundation of Guangdong Province (grant # 2016A030313067 [to HX])

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Collectively, the gut microbiota dysbiosis mediated by these specific bacterial populations may jointly promote the occurrence and progression of OA through multiple pathways such as regulating the hosts immune-inflammatory response, metabolic disorders, and oxidative stress, providing novel targets and strategies based on gut microbiota regulation for the prevention and treatment of OA

25 A nerve biopsy of the first known patient with the ACOX1 N237S mutation revealed a loss of Schwann cells

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