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While these relationships have been known for years, most literature has overlooked these findings as the predictable result of increased inflammation and oxidative stress similar to other biomarkers such as C-reactive protein (CRP)

The researchers exposed both naturally aging mice and mice genetically modified to age more rapidly to toxic chemotherapy drugs to induce additional stress on their cells

In the MSU-induced GA mouse model, complement C5a was secreted as a primary chemotactic signal for neutrophils (77), which could then bind to the G protein-coupled receptors to generate calcium ion flux (79), thereby mediating vesicle fusion with the cell membrane and promoting the secretion of exosomes coated with LTB4 (80)

Neonatal mice fed fat-deficient milk showed an extended postnatal cardiomyocyte proliferative window 34

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This stability is particularly significant for potential oral administration routes [4]