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Furthermore, LDL receptors become dysfunctional, increasing serum LDL-C, while inhibition of HDL-C concentration in the blood causes undesirable changes in lipid metabolism, producing hypercholesterolemia (Yousef et al

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Because Tirzepatide works on GLP-1 which slows digestion its side effects are very similar to other GLP-1 drugs: The good news tirzepatide does not cause hypoglycemia on its own

Occasionally, however, researchers do studies that provide evidence that allows us to test these competing hypotheses

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Elevated homocysteine is another risk factor in that assessment, although its causal effect on clotting is debated