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DOX is a highly toxic molecule and induces drug resistance due to the overexpression of ABCB1 efflux pump and other related proteins 127

Patel H, Chen J, Das KC, Kavdia M

The massive synthesis of pyruvate and the activation of the mTOR-HIF-1 pathway will further promote the aerobic glycolytic process, facilitating the TI process (134, 135)

There is no known pharmacological interaction from same-day administration

Doxorubicin has been reported to induce significant senescence in breast cancer cell lines, particularly promoting SA--gal expression in MCF7 cells line indicating a substantial impact of p53 on senescence induction [58]

When its pathways are well supported, adaptation is smoother