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*Gut Liver.* 14(2):153-167

Thus, regulation of mitophagy may be a valuable therapeutic option that targets the elimination of dysfunctional mitochondria while still providing sufficient energy to mitigate cellular damage, restore protein translation, and ultimately restore homeostasis ( FIGURE 1 3 Overview of mitophagy Mitophagy is a mitochondria-specific form of autophagy that selectively degrades damaged mitochondria through receptors (Figure 2)

It exists in both free and bound forms in the hypothalamus, limbic system, pituitary gland, and various peripheral organs, tissues, and fluids

Diclofenac, however, like all drugs that inhibit prostaglandin synthesis interfere with platelet function to some degree

This non-secretor genotype is a well-established risk factor for T1D and is associated with a different gut microbial community compared to secretor individuals (127, 128)

Medical supervision remains advisable for extended protocols