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p53 does not modulate ferroptosis via GSH export or altered electron transport chain function Activation of the p53p21 pathway can suppress ferroptosis by conserving intracellular GSH (Tarangelo et al, 2018)

Vodder School in Austria, the Foeldi School in Germany, and the University of Adelaide under Drs

These data showed that subchronic (7 to 14 days) exendin-4 or liraglutide administration in rodents resulted in hyperactivation of the HPA, disruption of glucocorticoid circadian secretion, hypertrophy of the adrenal gland, and dysregulation of pituitary-adrenal stress responses [77]

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