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Mitochondria can promote the progression of cysteine-deprivation-induced ferroptosis but not inhibit GPX4-induced ferroptosis [18]

Although the series had limited sample sizes, the findings are interesting

To compare the LPS- and semaglutide-induced treatment response, for each non-neuronal cell type, the Spearmans rho of the log fold-changes of the top 100 LPS-induced and the top 100 semaglutide-induced genes was computed

Negative feedback mechanisms from SOCS3 and PTP1B act to inhibit the receptor, ensuring that LEPR is not excessively activated

These mechanistic effects require further investigation before firm conclusions can be drawn