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Baertling, F, Rodenburg, RJ, Schaper, J, Smeitink, JA, Koopman, WJH, Mayatepek, E, Morava, E, Distelmaier, F 2014, A guide to diagnosis and treatment of Leigh syndrome, Journal of Neurology, Neurosurgery & Psychiatry , vol
The analysis covers the cellular mechanisms of intrinsic factor synthesis, its interaction with vitamin B12, and the complex absorption process that occurs in the terminal ileum
doi: 10.1210/jc.2006-1075 71 CanarisGJManowitzNRMayorGRidgwayEC
doi: 10.7453/gahmj.2013.011
Thus, acute Ca 2+ flux and persistent Ca 2+ dyshomeostasis are distinct cell death phenomena with the latter primarily affecting biosynthesis of ferroptosis substrates
After binding with PERK, TMAO selectively activates PERK-mediated UPR, thereby promoting metabolic dysfunction ( Of note, TMAO in the pathogenesis of Alzheimers disease (AD) indicated that PERK is not the only mechanism by which TMAO induces UPR
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