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Methodical progression provides clearer cause-and-effect understanding

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They found that TMAO promotes cell senescence in the CA3 region, damages the ultrastructure of synapse in the CA1 region, and increases oxidative stress in the hippocampus of mice

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Together, this highlights a need to further delineate differences in GLP1/GLP1 receptor signalling between species in future investigations in order to better understand the physiology versus pharmacology of the GLP1 system