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However, facing pathological stimuli such as protein aggregates, microglia are activated and adopt a pro-inflammatory phenotype, leading to the release of cytokines involving interleukin-1 (IL-1), interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-) to exacerbate neuronal impairment (Peruzzotti-Jametti et al., 2024

The changes were also observed in collagen deposition percentage, where methotrexate alone boosted it and more profound deposition was observed in carnitine-deficient MD-treated rats

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Lim, J.C., Grey, A.C., Zahraei, A., and Donaldson, P.J., Agedependent changes in glutathione metabolism pathways in the lens: New insights into therapeutic strategies to prevent cataract formation-A Review

Annals of Allergy, Asthma & Immunology, 82 (2), 186-191

There may be a threshold at which the neuroprotective functions of reactive astrocytes shift toward neurotoxic effects, acting as adversaries of neurons