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By integrating advances from plant biochemistry to molecular oncology, this review establishes an updated and mechanism-oriented framework for realizing SFNs compelling potential in cancer prevention and therapy through a precision medicine approach

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ChIP assays showed that, in contrast to WT ACTL6A, the ACTL6A HR-deletion mutant exhibited no restorative effect on the binding of NRF2 with the promoter of GCLC (Fig

Sestrin2 attenuates depressive-like behaviors and neuroinflammation in CUMS mice through inhibiting ferroptosis

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