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Briefly, these studies revealed that DSePA treatment (at concentrations which are non-toxic for normal cells) inhibited the growth of lung cancer cells by causing reductive stress (marked by the increase in the levels of GSH and GPx) followed by mitochondrial and endoplasmic reticulum-dependent apoptosis [92]

Reported Limitations and Considerations in GHK-Cu Research Gaps in the Current Evidence Base Despite a substantial pre-clinical record, GHK-Cu lacks large-scale human clinical trial data for most reported applications

doi: 10.1038/s41598-023-44607-4

It co-localizes with multiple hormones and neurotransmitters, suggesting broad regulatory functions