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However, the activity of L-DOPA in vitro (where it would need to be converted to dopamine to activate dopamine receptors), as well as the L-DOPAmediated bone loss and lack of effectiveness of D2 agonists on in vivo bone loss (Handa et al., 2019) indicate that the role of dopamine is more complex than just D2-signaling driving osteoclastogenesis and is likely affected by local changes in dopamine availability

10.1128/mBio.00632-19 170 HarnettN

This is a normal tissue response and not cause for concern

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