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J Neurol Neurosurg Psychiatry 61(6):565 Saenger S, Holtmann B, Nilges MR, Schroeder S, Hoeflich A, Kletzl H, Spooren W, Ostrowitzki S, Hanania T, Sendtner M, Metzger F (2012) Functional improvement in mouse models of familial amyotrophic lateral sclerosis by PEGylated insulin-like growth factor I treatment depends on disease severity

This design enables three distinct pharmacological actions in a single molecule: GLP-1 receptor agonism : The two conjugated GLP-1 analog peptides activate GLP-1R to produce appetite suppression, glucose-dependent insulin secretion, glucagon suppression, and delayed gastric emptying GIP receptor antagonism : The antibody component blocks GIP signaling, which may reduce fat storage and modify energy metabolism Extended half-life : The antibody backbone provides a half-life of approximately 21 days -- three times longer than any approved weekly incretin therapy -- enabling once-monthly dosing The GIP antagonism approach is the most distinctive feature

The bones: Hidden risks Bone is metabolically active and responds to incretin hormones, like GLP-1 and GIP, released in the intestines after eating to stimulate the pancreas to produce insulin

1999;277:R910-R916

Histological analysis reveals a reduction in fibrotic and necrotic regions, alongside improved villus morphology and myocardial structure

Thats going to help with (GLP-1 users) physical needs and mental needs, said Lyons Wyatt